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Department of Medicine

Department of Medicine

  Division of Pulmonary, Allergy and Critical Care Medicine



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photo Toru Nyunoya, MD

Associate Professor of Medicine

Email: nyunoyat@upmc.edu

Phone: 412-624-7280

Contact
Office: UPMC Montefiore Hospital, NW628
3459 Fifth Avenue
Pittsburgh, PA 15213
 
Phone: 412-624-7280
Fax: 412-624-1670
E-mail: nyunoyat@upmc.edu
Administrative Assistant:
Terri Heinrich
Email: heinrichta@upmc.edu
Phone: 412-624-7225
Fax: 412-624-1670
Education and Training
Education
MD, University of Tokushima School of Medicine, 1991
Training
Internship, Internal Medicine, University of Hawaii affiliated Hospitals, Honolulu, 1999
Residency, Internal Medicine, University of Hawaii affiliated Hospitals, Honolulu, 2001
Fellowship, Pulmonary and Critical Care, University of Iowa Hospitals and Clinics, 2004
Research Interest
Dr. Nyunoya’s research interests include the potential role of DNA repair in the development of Chronic Obstructive Pulmonary Disease (COPD) and novel modulators for smoking-induced COPD. He has an interest in identifying a natural product to protect against cigarette smoke-induced DNA damage and cytotoxicity
Publications
For my complete bibliography, Click Here.
Selected Publications:
Bruse, S., Moreau, M., Bromberg, Y., Jang, J.H., Wang, N., Ha, H., Picchi, M., Lin, Y., Langley, R.J., Qualls, C., Klensney-Tait, J., Zabner, J., Leng, S., Mao, J., Belinsky, S.A., Xing, J., Nyunoya, T. Whole exome sequencing identifies novel candidate genes that modify chronic obstructive pulmonary disease susceptibility. Human Genomics. 2016; 10: 1.
Jang, J.H., Bruse, S., Liu, Y., Duffy, V., Zhang, C., Oyamada, N., Randell, S., Matsumoto, A., Thompson, D.C., Lin, Y., Vasiliou, V., Tesfaigzi, Y., Nyunoya, T. Aldehyde dehydrogenase 3A1 protects airway epithelial cells from cigarette smoke-induced DNA damage and cytotoxicity. Free Radical Biology Medicine. 2014; 68: 80.
Jang, J.H., Bruse, S., Huneidi, S., Schrader, R.M., Monick, M.M., Lin, Y., Carter, A.B., Klingelhutz, A.J., Nyunoya T. Acrolein-exposed normal human lung fibroblasts in vitro: cellular senescence, enhanced telomere erosion, and degradation of Werner's syndrome protein. Environmental Health Perspective. 2014; 122: 955.